杀死一个PUA大师

不想被PUA的中科院物理所

2021-08-06 12:17:52

转自公众号：局部气候调查组

http://mp.weixin.qq.com/s?__biz=MzU0OTE1ODMxOA==&mid=2247511243&idx=1&sn=7032ca96c3aed536f57cd243ec715ef2

来源 | 局部气候调查组（ID：jubuqihou）

附件1:《参考资料》

[1] Walunas T L, Lenschow D J, Bakker C Y, et al. CTLA-4 can function as a negative regulator of T cell activation[J]. Immunity, 1994, 1(5): 405-413.

[2] Waterhouse P, Penninger J M, Timms E, et al. Lymphoproliferative disorders with early lethality in mice deficient in Ctla-4[J]. Science, 1995, 270(5238): 985-988.

[3] Leach D R, Krummel M F, Allison J P. Enhancement of antitumor immunity by CTLA-4 blockade[J]. Science, 1996, 271(5256): 1734-1736.

[4] Hodi F S, O'Day S J, McDermott D F, et al. Improved survival with ipilimumab in patients with metastatic melanoma[J]. New England Journal of Medicine, 2010, 363(8): 711-723.

[5] Postow M A, Chesney J, Pavlick A C, et al. Nivolumab and ipilimumab versus ipilimumab in untreated melanoma[J]. New England Journal of Medicine, 2015, 372(21): 2006-2017.

[6] Ishida, Y.; Agata, Y.; Shibahara, K.; Honjo, T. (1992). "Induced expression of PD-1, a novel member of the immunoglobulin gene superfamily, upon programmed cell death". The EMBO Journal. Wiley. 11 (11): 3887–3895.

[7] Dong H, Zhu G, Tamada K, Chen L (December 1999). "B7-H1, a third member of the B7 family, co-stimulates T-cell proliferation and interleukin-10 secretion". Nature Medicine. 5 (12): 1365–9.

附件2:《制作人员小憩》

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